If both of your parents and your grandparents suffered from hear disease then you may think you are also doomed to suffer from heart disease. There is good news, heredity can be a cause of heart disease, it is but one factor among may factors that must be taken into account when assessing your risk for heart disease. One recent study found that heredity accounts for less than 10 percent of a person’s risk for developing heart disease.
That leaves the other 90 percent of the heart disease causing factors that you may be able to do something about. If you are at risk because of heredity factor then modifying your life style and taking certain precautions could substantially reduce your risk of developing heart disease.
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Doctors cannot agree on the number one cause of heart disease, so you will have to evaluate the evidence yourself and determine your own risk/reward ratio. Smoking, obesity, and high cholesterol are usually in the forefront of any study.
The chemicals in cigarettes can damage artery walls, thereby making it easier for cholesterol deposits to build blood-blocking deposits on the artery walls. Smoking also makes platelets, the component of blood that causes clotting and carries oxygen, to be more active, thus increasing the risks of blood clots that cause heart attacks and storks.
A body needs cholesterol and can actually produce all it needs, so when we ingest foods high in cholesterols, like dairy and meat products, our bodies get a lot more cholesterol than they need. The body saves cholesterol instead of excreting it, and that cholesterol gets stored along the walls of the arteries. Too many cholesterol deposits lead to artery blockage and clots.
Having a large numbers of large HDL particles correlates with better health and it is commonly called “good cholesterol”. Having a large number of LDL particles in the blood is commonly called “bad cholesterol”. However, as today’s testing methods determine LDL (“bad”) and HDL (“good”) cholesterol separately, this simplistic view has become somewhat outdated.